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C. V. Mosby]
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C. V. Mosby
(221)
C.V. Mosby
(156)
C. V. Mosby]
(133)
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2001
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81
CARDIOPULMONARY SUPPORT AND PHYSIOLOGY - Neuropsychologic impairment after coronary bypass surgery: Effect of gaseous microemboli during perfusionist interventions/
Borger, M A
C.V. Mosby
2001
82
CARDIOPULMONARY SUPPORT AND PHYSIOLOGY - Percutaneous extracorporeal arteriovenous carbon dioxide removal improves survival in respiratory distress syndrome: A prospective randomized outcomes study in adult sheep/
Zwischenberger, J B
C.V. Mosby
2001
83
CARDIOPULMONARY SUPPORT AND PHYSIOLOGY - Protein kinase C isoform-dependent myocardial protection by ischemic preconditioning and potassium cardioplegia/
Lu, K
C.V. Mosby
2001
84
CARDIOPULMONARY SUPPORT AND PHYSIOLOGY - Proximal aortic dissection with coronary malperfusion: Presentation, management, and outcome/
Neri, E
C.V. Mosby
2001
85
CARDIOPULMONARY SUPPORT AND PHYSIOLOGY - P-selectin participates in cardiopulmonary bypass-induced inflammatory response in association with nitric oxide and peroxynitrite production - The anti-inflammatory effect of P-selectin monoclonal antibody on cardiopulmonary bypass (CPB)-induced inflammatory response was demonstrated in a rat CPB model by measuring P-selectin, inflammatory cytokines, nitrate/nitrite, nitrotyrosine, and respiratory index. P-selectin may help to augment CPB-induced inflammatory response in association with nitric oxide and peroxynitrite/
Hayashi, Yoshitaka
C.V. Mosby
2000
86
CARDIOPULMONARY SUPPORT AND PHYSIOLOGY - Similar neurobehavioral outcome after valve or coronary artery operations despite differing carotid embolic counts/
Neville, M J
C.V. Mosby
2001
87
CARDIOPULMONARY SUPPORT AND PHYSIOLOGY - Superior hepatic mitochondrial oxidation-reduction state in normothermic cardiopulmonary bypass/
Hashimoto, K
C.V. Mosby
2001
88
CARDIOPULMONARY SUPPORT AND PHYSIOLOGY - Temporal endothelin dynamics of the myocardial interstitium and systemic circulation in cardiopulmonary bypass/
Walker, C A
C.V. Mosby
2000
89
CARDIOPULMONARY SUPPORT AND PHYSIOLOGY - The effects of heparin and extracorporeal circulation on platelet counts and platelet microaggregation during cardiopulmonary bypass - Heparinization caused platelet activation, which increased existent platelet microaggregation ex vivo. During extracorporeal circulation, the loss of total circulating platelets was not associated with a significant change in the unaggregated platelet; count; existent platelet microaggregates were lost. Heparin may promote platelet loss by increasing microaggregate numbers/
Muriithi, Elijah W
C.V. Mosby
2000
90
CARDIOPULMONARY SUPPORT AND PHYSIOLOGY - The effects of retrograde cardioplegia technique on myocardial perfusion and energy metabolism: A magnetic resonance imaging and localized phosphorus 31 spectroscopy study in isolated pig hearts - Different methods of retrograde cardioplegia result in significant differences in distribution of cardioplegic solution/
Tian, Ganghong
C.V. Mosby
2000
91
CARDIOPULMONARY SUPPORT AND PHYSIOLOGY - The influence of human T lymphotropic virus type I infection on the outcome of cardiovascular surgery/
Masuda, H
C.V. Mosby
2000
92
CARDIOPULMONARY SUPPORT AND PHYSIOLOGY - The N-methyl-D-aspartate antagonist memantine has no neuroprotective effect during hypothermic circulatory arrest: A study in the chronic porcine model/
Rimpil�inen, J
C.V. Mosby
2001
93
CARDIOPULMONARY SUPPORT AND PHYSIOLOGY - The push-and-pull mechanism to scavenge redox-active transition metals: A novel concept in myocardial protection/
Karck, M
C.V. Mosby
2001
94
CARDIOPULMONARY SUPPORT AND PHYSIOLOGY - Tranexamic acid compared with high-dose aprotinin in primary elective heart operations: Effects on perioperative bleeding and allogeneic transfusions - We evaluated the effects of aprotinin and tranexamic acid on postoperative bleeding and the need for allogeneic transfusions in 1040 patients undergoing primary elective heart surgery. No clinical differences were found between the 2 drugs. Because tranexamic acid is 100 times cheaper than aprotinin, its use is preferable in this setting/
Casati, Valter
C.V. Mosby
2000
95
CARDIOTHORACIC TRANSPLANTATION - Adenovirus-mediated genetic manipulation of the myocardial b-adrenergic signaling system in transplanted hearts - Ex vivo manipulation of b-adrenergic receptor signaling offers a novel form of gene therapy to enhance myocardial function. Experimental cardiac allografts perfused with transgenes encoding either the human b2-adrenergic receptor or a peptide inhibitor of b-adrenergic receptor desensitization had significantly improved baseline and agonist-mediated LV performance compared with that found in controls/
Shah, Ashish S
C.V. Mosby
2000
96
CARDIOTHORACIC TRANSPLANTATION - Donor heart preservation with a novel hyperpolarizing solution: Superior protection compared with University of Wisconsin solution/
Hoenicke, E M
C.V. Mosby
2000
97
CARDIOTHORACIC TRANSPLANTATION - Early detection of acute allograft rejection by linear and nonlinear analysis of heart rate variability/
Izrailtyan, I
C.V. Mosby
2000
98
CARDIOTHORACIC TRANSPLANTATION - Inhibition of angiotensin-converting enzyme by captopril: A novel approach to reduce ischemia-reperfusion injury after lung transplantation - A rat single-lung transplant model was used to evaluate the ability of captopril to ameliorate ischemia-reperfusion injury by scavenging free radicals. The addition of captopril to the preservation solution significantly improved post-transplantation lung function, edema, and airway pressure; however, this is not mediated by a reduction in lipid peroxidation/
Fischer, Stefan
C.V. Mosby
2000
99
CARDIOTHORACIC TRANSPLANTATION - Ischemic preconditioning with opening of mitochondrial adenosine triphosphate-sensitive potassium channels or Na+/H+ exchange inhibition: Which is the best protective strategy for heart transplants?/
Kevelaitis, E
C.V. Mosby
2001
100
CARDIOTHORACIC TRANSPLANTATION - Low-potassium dextran solution ameliorates reperfusion injury of the lung and protects surfactant function - In minipigs, lung preservation with EC solution followed by warm ischemia and reperfusion led to lung insufficiency, right heart failure, and impairment of surfactant metabolism. The use of LPD solution prevented right heart failure and protected surfactant function, thereby improving lung function in the reperfusion period/
Str�ber, Martin
C.V. Mosby
2000
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